Mission
A Silent Crisis
Antibiotics often fail not because bacteria are genetically resistant, but because they tolerate stress. In chronic conditions like Cystic Fibrosis and COPD, pathogens like M. abscessus and P. aeruginosa change their physical properties to survive host immunity and conventional antibiotics.
Most new antibiotics target genes assumed to be essential, but evolution lets bacteria mutate around them. We target something they cannot evolve away from: the mechanical cell state that lets them survive in the first place.
- Targeting global markets in health, nutrition, and industry
- Discovery of compounds driving mechanical change
- Harnessing mechanical change in evolutionarily disparate species
